Epigenetics in Biology and Medicine
Coordonnateur : Esteller Manel
Anomalous epigenetic patterns touch many areas of study including biomedical, scientific, and industrial. With perspectives from international experts, this resource offers an all-inclusive overview of epigenetics, which bridge DNA information and function by regulating gene expression without modifying the DNA sequence itself.
Epigenetics, in its most basic form, means heredity is not the sole determining factor in disease development. Rather, environmental and dietary factors can trigger a gene to behave in an unintended way, while the gene itself remains unchanged.
Epigenetics in Biology and Medicinediscusses cell biology and epigenetics? role in human, animal, and plant diseases, including distortions in DNA methylation. The text also covers histone modifications, and epigenomics, as well as dietary and environmental impacts on epigenetic changes.
Addresses These Important Questions:
Represents a Landmark Publication in this Cutting-Edge Field
Dr. Manel Esteller, the editor of the volume, is a leading expert in molecular genetics of endometrial carcinoma. Under his editorial guidance, this publication goes beyond heredity to explain the crucial role of epigenetics in plants and physical and psychological diseases.
This text is applicable to a wide range of researchers, including those invested in the applications of cell biology, those involved in disease research and deciding genetic patterns, and those coalitions concerned with the impact of epigenetics and possible cures. As a result, it has the potenti
Date de parution : 08-2008
15.6x23.4 cm
Date de parution : 10-2019
15.6x23.4 cm
Mots-clés :
DNA Methylation; PRC2; cancer DNA methylation; ICF Syndrome; epigenetic drug combinations; PRC2 Complex; biomedical research; Acetyl K16 H4; PRC1 Complex; PRC1; DNA Methylation Pattern; DNA Hypomethylation; Aberrant DNA Methylation; DNA Demethylation; Compare DNA Methylation; Histone Code; DNA Methylation Profile; H3 Modification; DNA Methylation Marker; Hypermethylated Tumor Suppressor Genes; DNA Hypermethylation; Cell Cycle Progression; Histone Marks; Histone H4; FLC; FLC Expression; PcG Target; Hypomethylating Agents