Autophagy in Immune Response: Impact on Cancer Immunotherapy Breaking Tolerance to Anti-Cancer Cell-Mediated Immunotherapy Series
Coordonnateur : Chouaib Salem
Autophagy in Immune Response: Impact on Cancer Immunotherapy focuses on the status and future directions of autophagy with respect to different aspects of its interaction with the immune system and immunotherapy. The book takes scientific research in autophagy a step further by presenting reputable information on the topic and offering integrated content with advancements in autophagy, from cell biology and biochemical research, to clinical treatments. This book is a valuable source for cancer researchers, oncologists, graduate students and several members of biomedical field who are interested in learning more on the relationship between autophagy and immunotherapies.
- Presents updated knowledge on autophagy at the basic level and its potential use in cancer treatment
- Offers the first book to cover autophagy at the interface of cell biology, immunology and tumor biology
- Provides a wealth of information on the topic in a coherent and comprehensive collection of contributions by world renowned scientists and investigators
Date de parution : 05-2020
Ouvrage de 242 p.
19x23.3 cm
Thème d’Autophagy in Immune Response: Impact on Cancer Immunotherapy :
Mots-clés :
“Autophagy-related (ATG) genes; Antibody; Anticancer immunity; Antigen presentation and processing; ATG genes; ATP; Autophagy; Autophagy-mediated immunoadjuvant signals; Calreticulin; Cancer; Cancer immunosurveillance; Cancer immunotherapy; Cancer progression; CD4 T cells; Cell death; Complement; Concanavalin A; Cross talks; Cross-presentation; Cytokine; Cytotoxic T cells; Damage associated molecular patterns (DAMPs); Endoplasmic reticulum; Exosome biogenesis; Extracellular vesicles (EVs); Galectins; Glioblastoma; Glioma; HMGB1; Homeostasis; Hypoxia; Immune cells; Immune escape; Immune evasion; Immune resistance; Immune response; Immunity; Immunoglobulin; Immunosuppression; Immunotherapy; Infection; Inflammation; Interleukine; Lectins; MAP1LC3 conjugation machinery; MHC-I; MHC-II; Natural killer cells; Oncolytic viruses; Pathology; Protein secretion; Quality control; Resistance; RKIP; Therapeutics; Tumor; Tumor cells; Tumor microenvironment; Type 1 interferon (IFN Type 1); Yin Yang 1 (YY1)