Ischemic and Traumatic Brain and Spinal Cord Injuries Mechanisms and Potential Therapies
Auteur : Farooqui Akhlaq A.
Ischemic and Traumatic Brain and Spinal Cord Injuries: Mechanisms and Potential Therapies presents readers with comprehensive and cutting-edge information on molecular mechanisms, including the signal transduction processes associated with neurodegeneration and neuroprotection in ischemic, spinal cord, severe and mild brain injuries. The book also covers the molecular mechanisms of drugs used for the treatment of neurotraumatic disease. Chapters are organized by molecular aspects and neuroprotective strategies by disease, including ischemic injury, spinal cord injury, traumatic brain injury, and chronic traumatic encephalopathy.
The book integrates and consolidates knowledge on neurotraumatic diseases, with the hope of bringing forth more dramatic advances and developments, not only on molecular mechanisms, but also on the causes of, and treatments for, neurotraumatic diseases.
Researchers, Practitioners and advanced graduate students in Neuroscience, Neurology, and Pharmacology
- Offers a clearly written, logically organized, comprehensive overview on the molecular aspects of risk factors, pathogenesis, biomarkers, and therapeutic strategies for ischemic, spinal cord, severe and mild brain injuries
- Provides cutting-edge research information on the signal transduction processes associated with neurodegeneration and neuroprotection in ischemic, spinal cord, severe and mild brain injuries
- Presents chapters that are organized by molecular aspects and neuroprotective strategies by disease, including ischemic injury, spinal cord injury, traumatic brain injury, and chronic traumatic encephalopathy
Date de parution : 03-2018
Ouvrage de 484 p.
15x22.8 cm
Thème d’Ischemic and Traumatic Brain and Spinal Cord Injuries :
Mots-clés :
Acute neurodegeneration; Alzheimer's disease; Amyotrophic lateral sclerosis; And cognitive deficits; And seizures; Apoptosis; Astrocytes; Blood flow; CTE; Calcium influx; Chemokines; Chronic neurodegeneration; Chronic traumatic encephalopathy; Clot formation; Concussion; Curcumin; Cyclosporin; Cytokines; Dementia; Demyelination; Excitotoxicity; Glial scar; Glutamate; Hemorrhagic stroke; Huntington's disease; Hyperphosphorylated tau and TDP-43; Inflammation; Ischemic core; Ischemic stroke; Methylprednisolone sodium succinate; Microglia; Minocycline; Monosialoganglioside; NNZ-2566; Necrosis; Neuroinflammation; Neuronal dysfunction; Neuroprotectants; Neurotrophins; Oxidative stress; Parkinson's disease; Penumbra; Polyethylene glycol; Progesterone; Propranolol; ROS; Reactive oxygen species; Repetitive TBI; Repetitive mild TBI; Riluzole; Severe TBI; Sodium channels; Spinal cord injury; Statins stem cell therapy; Stroke; Tissue plasminogen activator; Traumatic brain injury; Traumatic brain injury (TBI)